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slc25a39 is necessary for mitochondrial glutathione import in mammalian cells DLAT Sustains Redox Homeostasis and Prevent Colorectal Cancer from Ferroptosis by Regulating SLC25A39-Mediated Mitochondrial Glutathione Transport
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slc25a39 is necessary for mitochondrial glutathione import in mammalian cells DLAT Sustains Redox Homeostasis and Prevent Colorectal Cancer from Ferroptosis by Regulating SLC25A39-Mediated Mitochondrial Glutathione Transport
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Mitochondrial GSH depletion sensitizes cells to BSO treatment a, Scheme Download Scientific Diagram PDF) Mitochondrial Glutathione Import Enables Breast Cancer Metastasis via Integrated Stress Response Signaling The glutathione system maintains the thiol redox balance in the mitochondria of fission yeast ScienceDirect Frontiers Regulation of Heme Synthesis by Mitochondrial Homeostasis Proteins SLC25A39 is necessary for mitochondrial glutathione import in mammalian cells Nature Use of Genetically Encoded Tools to Interrogate Mechanisms of Glutathione Homeostasis from Mitochondria to Mice
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slc25a39 is necessary for mitochondrial glutathione import in mammalian cells DLAT Sustains Redox Homeostasis and Prevent Colorectal Cancer from Ferroptosis by Regulating SLC25A39-Mediated Mitochondrial Glutathione Transport
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